Phagocytosis of bacteria and bacterial pathogenicity / edited by Joel D. Ernst and Olle Stendahl.

Other titles
  • Phagocytosis of Bacteria & Bacterial Pathogenicity.
  • Cambridge : Cambridge University Press 2006
Physical description
1 online resource (ix, 285 pages) : digital, PDF file(s).
  • 9780511541513 (ebook)
  • 9780521845694 (hardback)
  • Title from publisher's bibliographic system (viewed on 05 Oct 2015).
  • Available in electronic full text to members of the University via the Library web catalogue.
  • Introduction / Olle Stendahl -- Phagocytosis: receptors and biology / Wouter L.W. Hazenbos and Eric J. Brown -- Receptor-initiated signal transduction during phagocytosis / Kassidy K. Huynh and Sergio Grinstein -- Life, death, and inflammation: manipulation of phagocyte function by Helicobacter pylori / Lee-Ann H. Allen -- Phagocytosis of Streptococcus pneumoniae / Dominic L. Jack, David H. Dockrell, Stephen B. Gordon, and Robert C. Read -- Yersinia inhibiton of phagocytosis / Maria Fallman and Anna Gustavsson -- Listeria invasion and spread in non-professional phagocytes / Frederick S. Southwick -- Mycobacterium tuberculosis: mechanisms of phagocytosis and intracellular survival / Joel D. Ernst and Andrea Wolf.
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Related item
  • text
  • English
  • This book provides up-to-date information on the crucial interaction of pathogenic bacteria and professional phagocytes, the host cells whose purpose is to ingest, kill, and digest bacteria in defense against infection. The introductory chapters focus on the receptors used by professional phagocytes to recognize and phagocytose bacteria, and the signal transduction events that are essential for phagocytosis of bacteria. Subsequent chapters discuss specific bacterial pathogens and the strategies they use in confronting professional phagocytes. Examples include Helicobacter pylori, Streptococcus pneumoniae, and Yersinae, each of which uses distinct mechanisms to avoid being phagocytosed and killed. Contrasting examples include Listeria monocytogenes and Mycobacterium tuberculosis, which survive and replicate intracellularly, and actually cooperate with phagocytes to promote their entry into these cells. Together, the contributions in this book provide an outstanding review of current knowledge regarding the mechanisms of phagocytosis and how specific pathogenic bacteria avoid or exploit these mechanisms.

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